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Other molecular mechanisms behind the unfavourable effects of AAS on the lipoprotein profile have not been well investigated. Substituents at neighbouring atoms they are retained for the corresponding enzyme names. This may be an important anti-inflammatory mechanism as it allows corticosteroids to switch off the ongoing production of inflammatory proteins after the inflammatory gene has been activated. The findings may be limited by a lack of clarity around where the patients were in their disease course when randomized.
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